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Natalia V Oleinik Selected Research

formyltetrahydrofolate dehydrogenase

1/200910-formyltetrahydrofolate dehydrogenase-induced c-Jun-NH2-kinase pathways diverge at the c-Jun-NH2-kinase substrate level in cells with different p53 status.
11/2005Cancer cells activate p53 in response to 10-formyltetrahydrofolate dehydrogenase expression.
6/2003Ectopic expression of 10-formyltetrahydrofolate dehydrogenase in A549 cells induces G1 cell cycle arrest and apoptosis.
5/200210-formyltetrahydrofolate dehydrogenase, one of the major folate enzymes, is down-regulated in tumor tissues and possesses suppressor effects on cancer cells.

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Natalia V Oleinik Research Topics

Disease

9Neoplasms (Cancer)
09/2014 - 05/2002
1Carcinoma (Carcinomatosis)
09/2014
1Lung Neoplasms (Lung Cancer)
09/2014
1Neoplasm Metastasis (Metastasis)
09/2014
1Hepatocellular Carcinoma (Hepatoma)
02/2011

Drug/Important Bio-Agent (IBA)

4formyltetrahydrofolate dehydrogenaseIBA
01/2009 - 05/2002
3Folic Acid (Vitamin M)FDA LinkGeneric
09/2014 - 05/2002
3Proteins (Proteins, Gene)FDA Link
01/2013 - 05/2002
3EnzymesIBA
07/2010 - 05/2002
2Messenger RNA (mRNA)IBA
02/2011 - 05/2002
1Actin Depolymerizing Factors (Cofilins)IBA
09/2014
1rho GTP-Binding Proteins (rho GTP-Binding Protein)IBA
09/2014
1Glycine N-Methyltransferase (Glycine Sarcosine N-Methyltransferase)IBA
01/2013
1DNA (Deoxyribonucleic Acid)IBA
02/2011
1hydrogen sulfite (bisulfite)IBA
02/2011
1Leucovorin (Folinic Acid)FDA Link
07/2006